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Volume 61, Issue 3, Pages 348-358 (1 February 2007)


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The Prevalence and Correlates of Eating Disorders in the National Comorbidity Survey Replication

James I. HudsonaCorresponding Author Informationemail address, Eva Hiripib, Harrison G. Pope Jr.a, Ronald C. Kesslerb

Received 7 October 2005; received in revised form 10 February 2006; accepted 29 March 2006. published online 03 July 2006.

Background

Little population-based data exist on the prevalence or correlates of eating disorders.

Methods

Prevalence and correlates of eating disorders from the National Comorbidity Replication, a nationally representative face-to-face household survey (n = 9282), conducted in 2001–2003, were assessed using the WHO Composite International Diagnostic Interview.

Results

Lifetime prevalence estimates of DSM-IV anorexia nervosa, bulimia nervosa, and binge eating disorder are .9%, 1.5%, and 3.5% among women, and .3% .5%, and 2.0% among men. Survival analysis based on retrospective age-of-onset reports suggests that risk of bulimia nervosa and binge eating disorder increased with successive birth cohorts. All 3 disorders are significantly comorbid with many other DSM-IV disorders. Lifetime anorexia nervosa is significantly associated with low current weight (body-mass index <18.5), whereas lifetime binge eating disorder is associated with current severe obesity (body-mass index ≥40). Although most respondents with 12-month bulimia nervosa and binge eating disorder report some role impairment (data unavailable for anorexia nervosa since no respondents met criteria for 12-month prevalence), only a minority of cases ever sought treatment.

Conclusions

Eating disorders, although relatively uncommon, represent a public health concern because they are frequently associated with other psychopathology and role impairment, and are frequently under-treated.

a Department of Psychiatry, Harvard Medical School and Biological Psychiatry Laboratory, McLean Hospital, Belmont Massachusetts

b Department of Health Care Policy, Harvard Medical School, Cambridge Massachusetts.

Corresponding Author InformationAddress reprint requests to James I. Hudson, M.D., Biological Psychiatry Laboratory, McLean Hospital, 115 Mill Street, Belmont, MA 02478

PII: S0006-3223(06)00474-4

doi:10.1016/j.biopsych.2006.03.040


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